Jun 12, 2014 - link between somatosensation and motor control, I will report on how FD patients use sensory tricks to overcome their motor deficits and outline ...
JOURNAL OF SPORT & EXERCISE PSYCHOLOGY Volume 36 • Supplement • June 2014
North American Society for the Psychology of Sport and Physical Activity
Minneapolis, Minnesota June 12–14, 2014
Contents Keynotes and Lectures. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . S1 Symposia. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . S6 Free Communications: Verbal and Poster Motor Learning and Control. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . S17 Developmental Perspectives: Motor Control/Coordination/Rehabilitation. . . . . . . . . . . . . . . . . . . . . S59 Sport and Exercise Psychology. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . S79 Amendments to the Current Supplement, 2014 . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . S122 Author Index. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . S123
The Journal of Sport & Exercise Psychology is an official publication of the North American Society for the Psychology of Sport and Physical Activity.
Keynotes and Lectures S3
patients while walking poses a series of challenges. This presentation will describe how the use of a virtual reality gait paradigm in combination with functional neuroimaging, as well as ambulatory electroencephalography, have been able to investigate the neural correlates of FOG and, in turn, shed light on the processes underlying motor control in the brain.
Early Career Distinguished Scholar Lecture The reemergence of exploratory factor analysis in sport, exercise, and performance psychology Nicholas D. Myers; University of Miami, USA Over a century ago, Spearman (1904) articulated what has become to be known as exploratory factor analysis (EFA). Asparouhov and Muthén (2009) introduced exploratory structural equation modeling (ESEM) as a methodology that integrates the relative advantages of both EFA and confirmatory factor analysis (CFA) within the general structural equation model. Exploratory factor analysis has reemerged in sport, exercise, and performance psychology, in part because it has recently been integrated within ESEM (e.g., Morin & Maïano, 2011; Myers, Chase, Pierce, & Martin, 2011). Over 75 years ago, Holzinger and Swineford (1937) articulated what has become to be known as the bifactor model. The bifactor model has a general factor, group factors, and a pattern (or “loading”) matrix with a bifactor structure. Exploratory bifactor analysis (EBFA; Jennrich & Bentler, 2011, 2012) is EFA with a bifactor rotation criterion. There is reason to believe that (E)BFA is poised to emerge as a useful tool in sport, exercise, and performance psychology (e.g., Myers, Martin, Ntoumanis, Celimli, & Bartholomew, in press). The first purpose of this presentation is to provide a brief and general case for the possible conceptual utility of the bifactor model in sport, exercise, and performance psychology. The case for the first purpose relies on the observation that theory-based scales in sport, exercise, and performance psychology often are developed to measure a general continuous latent construct along with several more narrowly defined continuous latent subdomains (Tenenbaum, Eklund, & Kamata, 2012). The second purpose of this presentation is to demonstrate how exploratory (EBFA) and confirmatory (CBFA) forms of the bifactor model can be compared with each other and with more commonly used factor models in sport, exercise, and performance psychology within a substantive-methodological synergy format. The substantive focus is the consideration of the bifactor model for both the Psychological Need Thwarting Scale (Bartholomew, Ntoumanis, Ryan, & Thøgersen-Ntoumani, 2011) and the Physical Education Teaching Self-Efficacy Scale (Humphries, Hebert, Daigle, & Martin, 2012).
Senior Lecturers Developmental Perspectives: Motor Control/Coordination/Rehabilitation Proprioception and motor control across the life span Juergen Konczak; University of Minnesota, USA Proprioceptive afferent signals are essential for the reflexive control of muscle tone, for postural and volitional motor control. They also form the basis for our sense of body awareness and, when combined with tactile information during active touch, give rise to the haptic perception of our surrounds. Although the neurophysiology of proprioception is well understood, we know surprisingly little about how proprioceptive function develops during the life span and how it is altered by disease. In addition, we do not fully understand how proprioceptive information is integrated with information from other senses and mapped onto motor commands. In this talk I will present a series of studies from my lab and the work of others that investigated proprioceptive and haptic function in typically developing children and in aging. They highlight that proprioception and haptic perception continue to develop well into adolescence. They also demonstrate that haptic accuracy is quite resilient to aging. In a second part of this talk, I will report on how proprioception is affected in patients with dysfunction of the cerebellum or the basal ganglia, two neural structures known to be important for motor control and learning. I will highlight that basal ganglia–related diseases, such as Parkinson’s disease and focal dystonia (FD), show tactile, proprioceptive, and haptic deficits while kinesthesia remains unimpaired in cerebellar patients (Konczak et al., 2012; Maschke, Gomez, Tuite, & Konczak, 2003). To underline the intricate link between somatosensation and motor control, I will report on how FD patients use sensory tricks to overcome their motor deficits and outline the mechanism behind them (Konczak & Abbruzzese, 2013). I will argue that many of the observable motor deficits in basal ganglia disease have in fact a somatosensory origin.
